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Details for phenotype: regulation of sensory perception of pain

EndoNet ID: ENP00216

Name

regulation of sensory perception of pain

General information

This phenotype is not pathologic

Links to other resources

GO
MeSH term 68058748

Phenotype triggers

  • normal activity of PRLR
    in brain
    • Prolactin has been shown to have analgesic effects that can be mimicked by a number of central nervous system neurotransmitters. [1]
  • normal activity of FTS receptor
    in central_nerve_system_element
    • A series of experiments showing the hyperalgesic actions of thymulin in the nervous system. [2]
    • Thymulin may be acting directly or indirectly on nerve terminals of the CNS. PGE2 might be a key mediator in these actions. [2]
    • The attenuation of hyperalgesia by high doses of thymulin involve the downregulation of proinflammatory cytokines. [2]
Reference