Details for anatomical structure: dendritic cell in lymphoid tissues

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EndoNet ID: ENC00228

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Synonyms

dendritic cell in lymphoid tissues, , Macrophagocytus stabilis

Links to other resources

Cytomer

cy0011307

Larger structures

Substructures

Secreted hormones

Hormone: pentraxin 3
- Production of the soluble pattern recognition receptor PTX3 by myeloid, but not plasmacytoid, dendritic cells. [1]
- PTX3 is produced by a variety of cells and tissues, most notably dendritic cells and macrophages, in response to Toll-like receptor (TLR) engagement and inflammatory cytokines. [2]
- PTX3 is made by diverse cell types, most prominently endothelial cells, macrophages and dendritic cells, in response to primary inflammatory signals (e.g. interleukin-1 (IL-1), tumour necrosis factor (TNF), lipopolysaccharide (LPS)). [3]
Hormone: IL-18
Hormone: MDC
Hormone: MIP-3 beta
Hormone: PARC
Hormone: BAFF
Hormone: IL-12
- DC produce high levels of IL-12, the major cytokine responsible for the generation of inflammatory Th1 cells. [4]
Influenced by:
- biliary glycoprotein 1
  in neutrophil_granulocyte
  451 Evidence in mice suggests that biliary glycoprotein activation on murine dendritic cells causes IL-6 and IL-12 release. 1410 [5]
Hormone: PD-L1
Hormone: TNF-alpha
Hormone: IL-1 beta
Hormone: interleukin 6
Hormone: IFN-alpha
- DCs express IFN-alpha, IFN-beta, IL-28, IL-29, TNF-alpha and the chemokines CCL5 and CXCL10 after herpes simplex virus 1 (HSV-1) infection. [6]
Influenced by:
- TLR3
  in dendritic_cell_in_lymphoid_tissues
  TLR3, TLR7, and TLR9 can induce both IFN-alpha and IFN-beta. [7]
- TLR7
  in dendritic_cell_in_lymphoid_tissues
- TLR9
  in dendritic_cell_in_lymphoid_tissues
Hormone: IFN-beta
Influenced by:
- TLR3
  in dendritic_cell_in_lymphoid_tissues
  TLR3, TLR7, and TLR9 can induce both IFN-alpha and IFN-beta. [7]
- TLR7
  in dendritic_cell_in_lymphoid_tissues
- TLR9
  in dendritic_cell_in_lymphoid_tissues
- TLR4
  in dendritic_cell_in_lymphoid_tissues
  TLR4 can lead to IFN-beta production. [7]
Hormone: IL-29
Hormone: RANTES
Hormone: IP-10
Hormone: IL-10
Hormone: IL-1 alpha
Hormone: IL-23
Hormone: SLAMF1 isoform 3
Hormone: IL-8
Influenced by:
- TLR3
  in dendritic_cell_in_lymphoid_tissues
  RNA, likely through secondary structure, is a potent host-derived activator of TLR3. Exposing cells human embryonic kidney 293 cells stably expressing TLR3 and containing a nuclear factor-kB-dependent luciferase reporter) to in vitro transcribed RNA resulted in a TLR3-dependent induction of luciferase activity and interleukin-8 secretion. [8]
Hormone: ICAM-1
- hey showed by flow cytometry and immunohistochemistry that ICAM-1 was widely expressed on different cell types, including tissue macrophages and dendritic cells. It was also expressed on nonhematopoietic cells, such as vascular endothelial cells, thymic and mucosal epithelial cells, and dermal fibroblasts. [9]
Hormone: LFA-3
- The presence of LFA-3 on dendritic cells is consistent with the specialised function of these cells in presenting antigen to effector T-cells. [10]

Receptors

Receptor: H3
Receptor: H1
Receptor: H2
Receptor: histamine H4 receptor
Receptor: TLR8
Receptor: TLR3
Influences:
- IFN-alpha
  
  TLR3, TLR7, and TLR9 can induce both IFN-alpha and IFN-beta. [7]
- IFN-beta
  
  TLR3, TLR7, and TLR9 can induce both IFN-alpha and IFN-beta. [7]
- IL-8
  
  RNA, likely through secondary structure, is a potent host-derived activator of TLR3. Exposing cells human embryonic kidney 293 cells stably expressing TLR3 and containing a nuclear factor-kB-dependent luciferase reporter) to in vitro transcribed RNA resulted in a TLR3-dependent induction of luciferase activity and interleukin-8 secretion. [8]
Receptor: TLR4
Influences:
- IFN-beta
  
  TLR4 can lead to IFN-beta production. [7]
Receptor: TLR6
Receptor: TLR9
Influences:
- IFN-alpha
- IFN-beta
Receptor: SIGLEC-7
Receptor: PAF-R
Receptor: TLR7
Influences:
- IFN-alpha
- IFN-beta
Receptor: LIR-1
Receptor: CD5
Receptor: CD72
Influences:
- IFN-gamma
  
  Activated NK cells express CD100 and produce soluble CD100. They interact with dendritic cells by several routs among which is the CD100-CD72 interaction. Such interaction lead to IFNgamma secretion which in turn activates other cells of the immune system. [11]
- SEMA4D
  
  Activated NK cells express CD100 and produce soluble CD100. They interact with dentritic cells by several routs among which is the CD100-CD72 interaction. Such interaction lead to soluble CD100 secretion which in turn activates other cells of the immune system. [11]
Receptor: Sphingosine 1-phosphate receptor 1
Induced phenotype:
- dendritic cell chemotaxis
  
  FTY720, a S1P receptor agonist, can severly hamper migration of dendritic cells to the lymph nodes suggesting additional mechanisms underlying the immunosuppressive effects of this spingosine analog. [12]
Receptor: Sphingosine 1-phosphate receptor 2
Receptor: Sphingosine 1-phosphate receptor 3
Induced phenotype:
- dendritic cell chemotaxis
  
  DCs mainly express S1PR1 when immature but on maturation upregulate S1PR3, which then seems to mediate a chemotatic response to S1P. [13]
- positive regulation of endocytosis
  
  S1P has been shown to increase endocytosis by mature DCs through S1PR3 engagement. [14]
  This S1P-mediated enhancement of endocytosis in mature DCs could be involved in the removal of bacteria during infection. [14]
- positive regulation of interleukin-1 beta production
  
  S1PR3 is essential for inducing the production of IL-1beta and tissue factor and for their dissemination into the lung circulation, thereby amplifying inflammation and coagulation. [15]
Receptor: Sphingosine 1-phosphate receptor 4
Induced phenotype:
- regulation of T cell differentiation
  
  S1P has been shown to modulate the polarizing functions of DCs on T cells when exposed to bacterial products. [16]
- regulation of leukocyte migration
  
  S1PR4 is dominantly expressed on mouse dendritic cells and has an important role in DC trafficking. [17]
- dendritic cell chemotaxis
  
  FTY720, a S1P receptor agonist, can severly hamper migration of dendritic cells to the lymph nodes suggesting additional mechanisms underlying the immunosuppressive effects of this spingosine analog. [12]
Receptor: Sphingosine 1-phosphate receptor 5
Induced phenotype:
- dendritic cell chemotaxis
  
  FTY720, a S1P receptor agonist, can severly hamper migration of dendritic cells to the lymph nodes suggesting additional mechanisms underlying the immunosuppressive effects of this spingosine analog. [12]
Receptor: Lysophosphatidic acid receptor 3
Induced phenotype:
- dendritic cell chemotaxis
  
  LPA3 activation induced chemotaxis of immature, but not mature, DCs. [18]
Receptor: Probable G-protein coupled receptor 132
Induced phenotype:
- regulation of innate immune response
  
  Innate immune responses of dendritic cells may be regulated via G2A in response to local fluctuations in lysolipids encountered during infection and inflammation. It is possible that LPC produced at sites of inflammation and from disintegrating apoptotic/necrotic cell membranes may be a novel “molecular pattern” recognized by G2A to modulate innate immune processes and the initiation and/or resolution of inflammatory responses. [19]
Receptor: CMKLR1
- We have previously described ChemR23 as a human orphan G protein–coupled receptor expressed in macrophages and immature DCs [20]
Induced phenotype:
- dendritic cell chemotaxis
  
  Human recombinant chemerin promoted in vitro migration of immature DCs whereas no chemotaxis of mature DCs was observed [20]
Receptor: CCR6
- human CCR6, a chemokine receptor preferentially expressed by immature dendritic cells and memory T cells. [21]
Induced phenotype:
- dendritic cell chemotaxis
  
  β-defensins may promote adaptive immune responses by recruiting dendritic and T cells to the site of microbial invasion through interaction with CCR6. [21]
  CC-chemokine receptor (CCR) 6 is the only known receptor for macrophage inflammatory protein (MIP)-3alpha, a CC chemokine chemotactic for lymphocytes and dendritic cells. [22]
Receptor: CCR7
- Chemokine receptor CCR7 is expressed on mature DC. [23]
Induced phenotype:
- dendritic cell chemotaxis
  
  Migration of DC to lymph nodes is regulated by chemokine receptor CCR7, expressed on mature DC, and the CCR7 ligands CCL19 and CCL21. [23]

Reference

EndoNet

The information resource about the endocrine network in human.

Details for anatomical structure: dendritic cell in lymphoid tissues

Synonyms

Links to other resources

Related structures

Larger structures

Substructures

Secreted hormones

Hormone: pentraxin 3 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_0_toolsBtn',{id:'j_idt91:0:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_0_j_idt99',{id:'j_idt91:0:j_idt99',target:'j_idt91:0:toolsBtn',dynamic:true});});

Hormone: IL-18 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_1_toolsBtn',{id:'j_idt91:1:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_1_j_idt99',{id:'j_idt91:1:j_idt99',target:'j_idt91:1:toolsBtn',dynamic:true});});

Hormone: MDC ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_2_toolsBtn',{id:'j_idt91:2:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_2_j_idt99',{id:'j_idt91:2:j_idt99',target:'j_idt91:2:toolsBtn',dynamic:true});});

Hormone: MIP-3 beta ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_3_toolsBtn',{id:'j_idt91:3:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_3_j_idt99',{id:'j_idt91:3:j_idt99',target:'j_idt91:3:toolsBtn',dynamic:true});});

Hormone: PARC ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_4_toolsBtn',{id:'j_idt91:4:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_4_j_idt99',{id:'j_idt91:4:j_idt99',target:'j_idt91:4:toolsBtn',dynamic:true});});

Hormone: BAFF ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_5_toolsBtn',{id:'j_idt91:5:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_5_j_idt99',{id:'j_idt91:5:j_idt99',target:'j_idt91:5:toolsBtn',dynamic:true});});

Hormone: IL-12 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_6_toolsBtn',{id:'j_idt91:6:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_6_j_idt99',{id:'j_idt91:6:j_idt99',target:'j_idt91:6:toolsBtn',dynamic:true});});

Influenced by:

Hormone: PD-L1 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_7_toolsBtn',{id:'j_idt91:7:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_7_j_idt99',{id:'j_idt91:7:j_idt99',target:'j_idt91:7:toolsBtn',dynamic:true});});

Hormone: TNF-alpha ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_8_toolsBtn',{id:'j_idt91:8:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_8_j_idt99',{id:'j_idt91:8:j_idt99',target:'j_idt91:8:toolsBtn',dynamic:true});});

Hormone: IL-1 beta ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_9_toolsBtn',{id:'j_idt91:9:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_9_j_idt99',{id:'j_idt91:9:j_idt99',target:'j_idt91:9:toolsBtn',dynamic:true});});

Hormone: interleukin 6 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_10_toolsBtn',{id:'j_idt91:10:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_10_j_idt99',{id:'j_idt91:10:j_idt99',target:'j_idt91:10:toolsBtn',dynamic:true});});

Hormone: IFN-alpha ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_11_toolsBtn',{id:'j_idt91:11:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_11_j_idt99',{id:'j_idt91:11:j_idt99',target:'j_idt91:11:toolsBtn',dynamic:true});});

Influenced by:

Hormone: IFN-beta ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_12_toolsBtn',{id:'j_idt91:12:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_12_j_idt99',{id:'j_idt91:12:j_idt99',target:'j_idt91:12:toolsBtn',dynamic:true});});

Influenced by:

Hormone: IL-29 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_13_toolsBtn',{id:'j_idt91:13:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_13_j_idt99',{id:'j_idt91:13:j_idt99',target:'j_idt91:13:toolsBtn',dynamic:true});});

Hormone: RANTES ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_14_toolsBtn',{id:'j_idt91:14:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_14_j_idt99',{id:'j_idt91:14:j_idt99',target:'j_idt91:14:toolsBtn',dynamic:true});});

Hormone: IP-10 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_15_toolsBtn',{id:'j_idt91:15:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_15_j_idt99',{id:'j_idt91:15:j_idt99',target:'j_idt91:15:toolsBtn',dynamic:true});});

Hormone: IL-10 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_16_toolsBtn',{id:'j_idt91:16:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_16_j_idt99',{id:'j_idt91:16:j_idt99',target:'j_idt91:16:toolsBtn',dynamic:true});});

Hormone: IL-1 alpha ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_17_toolsBtn',{id:'j_idt91:17:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_17_j_idt99',{id:'j_idt91:17:j_idt99',target:'j_idt91:17:toolsBtn',dynamic:true});});

Hormone: IL-23 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_18_toolsBtn',{id:'j_idt91:18:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_18_j_idt99',{id:'j_idt91:18:j_idt99',target:'j_idt91:18:toolsBtn',dynamic:true});});

Hormone: SLAMF1 isoform 3 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_19_toolsBtn',{id:'j_idt91:19:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_19_j_idt99',{id:'j_idt91:19:j_idt99',target:'j_idt91:19:toolsBtn',dynamic:true});});

Hormone: IL-8 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_20_toolsBtn',{id:'j_idt91:20:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_20_j_idt99',{id:'j_idt91:20:j_idt99',target:'j_idt91:20:toolsBtn',dynamic:true});});

Influenced by:

Hormone: ICAM-1 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_21_toolsBtn',{id:'j_idt91:21:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_21_j_idt99',{id:'j_idt91:21:j_idt99',target:'j_idt91:21:toolsBtn',dynamic:true});});

Hormone: LFA-3 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt91_22_toolsBtn',{id:'j_idt91:22:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt91_22_j_idt99',{id:'j_idt91:22:j_idt99',target:'j_idt91:22:toolsBtn',dynamic:true});});

Receptors

Receptor: H3 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_0_toolsBtn',{id:'j_idt139:0:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_0_j_idt145',{id:'j_idt139:0:j_idt145',target:'j_idt139:0:toolsBtn',dynamic:true});});

Receptor: H1 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_1_toolsBtn',{id:'j_idt139:1:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_1_j_idt145',{id:'j_idt139:1:j_idt145',target:'j_idt139:1:toolsBtn',dynamic:true});});

Receptor: H2 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_2_toolsBtn',{id:'j_idt139:2:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_2_j_idt145',{id:'j_idt139:2:j_idt145',target:'j_idt139:2:toolsBtn',dynamic:true});});

Receptor: histamine H4 receptor ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_3_toolsBtn',{id:'j_idt139:3:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_3_j_idt145',{id:'j_idt139:3:j_idt145',target:'j_idt139:3:toolsBtn',dynamic:true});});

Receptor: TLR8 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_4_toolsBtn',{id:'j_idt139:4:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_4_j_idt145',{id:'j_idt139:4:j_idt145',target:'j_idt139:4:toolsBtn',dynamic:true});});

Receptor: TLR3 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_5_toolsBtn',{id:'j_idt139:5:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_5_j_idt145',{id:'j_idt139:5:j_idt145',target:'j_idt139:5:toolsBtn',dynamic:true});});

Influences:

Receptor: TLR4 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_6_toolsBtn',{id:'j_idt139:6:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_6_j_idt145',{id:'j_idt139:6:j_idt145',target:'j_idt139:6:toolsBtn',dynamic:true});});

Influences:

Receptor: TLR6 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_7_toolsBtn',{id:'j_idt139:7:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_7_j_idt145',{id:'j_idt139:7:j_idt145',target:'j_idt139:7:toolsBtn',dynamic:true});});

Receptor: TLR9 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_8_toolsBtn',{id:'j_idt139:8:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_8_j_idt145',{id:'j_idt139:8:j_idt145',target:'j_idt139:8:toolsBtn',dynamic:true});});

Influences:

Receptor: SIGLEC-7 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_9_toolsBtn',{id:'j_idt139:9:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_9_j_idt145',{id:'j_idt139:9:j_idt145',target:'j_idt139:9:toolsBtn',dynamic:true});});

Receptor: PAF-R ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_10_toolsBtn',{id:'j_idt139:10:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_10_j_idt145',{id:'j_idt139:10:j_idt145',target:'j_idt139:10:toolsBtn',dynamic:true});});

Receptor: TLR7 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_11_toolsBtn',{id:'j_idt139:11:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_11_j_idt145',{id:'j_idt139:11:j_idt145',target:'j_idt139:11:toolsBtn',dynamic:true});});

Influences:

Receptor: LIR-1 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_12_toolsBtn',{id:'j_idt139:12:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_12_j_idt145',{id:'j_idt139:12:j_idt145',target:'j_idt139:12:toolsBtn',dynamic:true});});

Receptor: CD5 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_13_toolsBtn',{id:'j_idt139:13:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_13_j_idt145',{id:'j_idt139:13:j_idt145',target:'j_idt139:13:toolsBtn',dynamic:true});});

Receptor: CD72 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_14_toolsBtn',{id:'j_idt139:14:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_14_j_idt145',{id:'j_idt139:14:j_idt145',target:'j_idt139:14:toolsBtn',dynamic:true});});

Influences:

Receptor: Sphingosine 1-phosphate receptor 1 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_15_toolsBtn',{id:'j_idt139:15:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_15_j_idt145',{id:'j_idt139:15:j_idt145',target:'j_idt139:15:toolsBtn',dynamic:true});});

Induced phenotype:

Receptor: Sphingosine 1-phosphate receptor 2 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_16_toolsBtn',{id:'j_idt139:16:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_16_j_idt145',{id:'j_idt139:16:j_idt145',target:'j_idt139:16:toolsBtn',dynamic:true});});

Receptor: Sphingosine 1-phosphate receptor 3 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_17_toolsBtn',{id:'j_idt139:17:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_17_j_idt145',{id:'j_idt139:17:j_idt145',target:'j_idt139:17:toolsBtn',dynamic:true});});

Induced phenotype:

Receptor: Sphingosine 1-phosphate receptor 4 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_18_toolsBtn',{id:'j_idt139:18:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_18_j_idt145',{id:'j_idt139:18:j_idt145',target:'j_idt139:18:toolsBtn',dynamic:true});});

Induced phenotype:

Receptor: Sphingosine 1-phosphate receptor 5 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_19_toolsBtn',{id:'j_idt139:19:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_19_j_idt145',{id:'j_idt139:19:j_idt145',target:'j_idt139:19:toolsBtn',dynamic:true});});

Induced phenotype:

Receptor: Lysophosphatidic acid receptor 3 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_20_toolsBtn',{id:'j_idt139:20:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_20_j_idt145',{id:'j_idt139:20:j_idt145',target:'j_idt139:20:toolsBtn',dynamic:true});});

Induced phenotype:

Receptor: Probable G-protein coupled receptor 132 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_21_toolsBtn',{id:'j_idt139:21:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_21_j_idt145',{id:'j_idt139:21:j_idt145',target:'j_idt139:21:toolsBtn',dynamic:true});});

Induced phenotype:

Receptor: CMKLR1 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_22_toolsBtn',{id:'j_idt139:22:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_22_j_idt145',{id:'j_idt139:22:j_idt145',target:'j_idt139:22:toolsBtn',dynamic:true});});

Induced phenotype:

Receptor: CCR6 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_23_toolsBtn',{id:'j_idt139:23:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_23_j_idt145',{id:'j_idt139:23:j_idt145',target:'j_idt139:23:toolsBtn',dynamic:true});});

Induced phenotype:

Receptor: CCR7 ui-buttonPrimeFaces.cw('CommandButton','widget_j_idt139_24_toolsBtn',{id:'j_idt139:24:toolsBtn'}); $(function(){PrimeFaces.cw('OverlayPanel','widget_j_idt139_24_j_idt145',{id:'j_idt139:24:j_idt145',target:'j_idt139:24:toolsBtn',dynamic:true});});

Induced phenotype:

Hormone: pentraxin 3

Hormone: IL-18

Hormone: MDC

Hormone: MIP-3 beta

Hormone: PARC

Hormone: BAFF

Hormone: IL-12

Hormone: PD-L1

Hormone: TNF-alpha

Hormone: IL-1 beta

Hormone: interleukin 6

Hormone: IFN-alpha

Hormone: IFN-beta

Hormone: IL-29

Hormone: RANTES

Hormone: IP-10

Hormone: IL-10

Hormone: IL-1 alpha

Hormone: IL-23

Hormone: SLAMF1 isoform 3

Hormone: IL-8

Hormone: ICAM-1

Hormone: LFA-3

Receptor: H3

Receptor: H1

Receptor: H2

Receptor: histamine H4 receptor

Receptor: TLR8

Receptor: TLR3

Receptor: TLR4

Receptor: TLR6

Receptor: TLR9

Receptor: SIGLEC-7

Receptor: PAF-R

Receptor: TLR7

Receptor: LIR-1

Receptor: CD5

Receptor: CD72

Receptor: Sphingosine 1-phosphate receptor 1

Receptor: Sphingosine 1-phosphate receptor 2

Receptor: Sphingosine 1-phosphate receptor 3

Receptor: Sphingosine 1-phosphate receptor 4

Receptor: Sphingosine 1-phosphate receptor 5

Receptor: Lysophosphatidic acid receptor 3

Receptor: Probable G-protein coupled receptor 132

Receptor: CMKLR1

Receptor: CCR6

Receptor: CCR7